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A. In cancer cells, excessive production of BCL-2 sequesters and

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Last updated 17 abril 2025
A. In cancer cells, excessive production of BCL-2 sequesters and
A. In cancer cells, excessive production of BCL-2 sequesters and
Targeting BCL-2 regulated apoptosis in cancer
A. In cancer cells, excessive production of BCL-2 sequesters and
IJMS, Free Full-Text
A. In cancer cells, excessive production of BCL-2 sequesters and
Downregulation of autophagy by Bcl-2 promotes MCF7 breast cancer cell growth independent of its inhibition of apoptosis
A. In cancer cells, excessive production of BCL-2 sequesters and
Bcl‐2 proteins in development, health, and disease of the hematopoietic system - Kollek - 2016 - The FEBS Journal - Wiley Online Library
A. In cancer cells, excessive production of BCL-2 sequesters and
Biomolecules, Free Full-Text
A. In cancer cells, excessive production of BCL-2 sequesters and
Targeting BCL-2 regulated apoptosis in cancer
A. In cancer cells, excessive production of BCL-2 sequesters and
Cancers, Free Full-Text
A. In cancer cells, excessive production of BCL-2 sequesters and
IJMS, Free Full-Text
A. In cancer cells, excessive production of BCL-2 sequesters and
A) Bcl-2 family member effector proteins (Bax and Bak) oligomerize and
A. In cancer cells, excessive production of BCL-2 sequesters and
Mechanisms of BCL-2 family proteins in mitochondrial apoptosis
A. In cancer cells, excessive production of BCL-2 sequesters and
Cell Survival and Cell Death at the Intersection of Autophagy and Apoptosis: Implications for Current and Future Cancer Therapeutics
A. In cancer cells, excessive production of BCL-2 sequesters and
A delicate balance – The BCL-2 family and its role in apoptosis, oncogenesis, and cancer therapeutics - ScienceDirect
A. In cancer cells, excessive production of BCL-2 sequesters and
Models for the pro-survival activity of Bcl-2 (A) Recent data
A. In cancer cells, excessive production of BCL-2 sequesters and
Anti-apoptotic BCL-2 regulation by changes in dynamics of its long unstructured loop
A. In cancer cells, excessive production of BCL-2 sequesters and
BCL-2 family members differ in their dependence on proapoptotic
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