Frontiers Deletion of MicroRNA-144/451 Cluster Aggravated Brain Injury in Intracerebral Hemorrhage Mice by Targeting 14-3-3ζ
Por um escritor misterioso
Last updated 11 novembro 2024
Microglial SLC25A28 Deficiency Ameliorates the Brain Injury After Intracerebral Hemorrhage in Mice by Restricting Aerobic Glycolysis
The antioxidant effects of melatonin after intracerebral hemorrhage in rats
Neuroprotective effects of hydrogen inhalation in an experimental rat intracerebral hemorrhage model
Frontiers Deletion of MicroRNA-144/451 Cluster Aggravated Brain Injury in Intracerebral Hemorrhage Mice by Targeting 14-3-3ζ
PDF) Modulation of Heme-Induced Inflammation Using MicroRNA-Loaded Liposomes: Implications for Hemolytic Disorders Such as Malaria and Sickle Cell Disease
Microglial SLC25A28 Deficiency Ameliorates the Brain Injury After Intracerebral Hemorrhage in Mice by Restricting Aerobic Glycolysis
Microglial SLC25A28 Deficiency Ameliorates the Brain Injury After Intracerebral Hemorrhage in Mice by Restricting Aerobic Glycolysis
Microglial SLC25A28 Deficiency Ameliorates the Brain Injury After Intracerebral Hemorrhage in Mice by Restricting Aerobic Glycolysis
Frontiers MiR-451a and let-7i-5p loaded extracellular vesicles attenuate heme-induced inflammation in hiPSC-derived endothelial cells
Frontiers Role and regulatory mechanism of microRNA mediated neuroinflammation in neuronal system diseases
PDF) Modulation of Heme-Induced Inflammation Using MicroRNA-Loaded Liposomes: Implications for Hemolytic Disorders Such as Malaria and Sickle Cell Disease
Yongan Sun's research works Beijing Medical University, Beijing and other places
Neuroprotective effects of hydrogen inhalation in an experimental rat intracerebral hemorrhage model
Frontiers Role and regulatory mechanism of microRNA mediated neuroinflammation in neuronal system diseases
Frontiers MicroRNAs modulate neuroinflammation after intracerebral hemorrhage: Prospects for new therapy
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