How SARS-CoV-2 hijacks human cells to evade i

UC San Diego School of Medicine researchers discovered one way in which SARS-CoV-2 hijacks human cell machinery to blunt the immune response, allowing it to establish infection, replicate and cause disease.

Molecular mechanisms involved in pathogenicity of SARS-CoV-2: Immune evasion and implications for therapeutic strategies - ScienceDirect

SARS-CoV-2 hijacks p38β/MAPK11 to promote virus replication

The battle between host and SARS-CoV-2: Innate immunity and viral evasion strategies: Molecular Therapy

SARS-CoV-2 hijacks p38β/MAPK11 to promote virus replication

Tight junction protein occludin is an internalization factor for SARS-CoV-2 infection and mediates virus cell-to-cell transmission

How Coronavirus Hijacks Your Cells - The New York Times

SARS-CoV-2 induces double-stranded RNA-mediated innate immune responses in respiratory epithelial-derived cells and cardiomyocytes

Frontiers SARS-CoV-2 and the Host Cell: A Tale of Interactions

How Coronavirus Hijacks Your Cells - The New York Times

Human models for COVID‐19 research - Woodall - 2021 - The Journal of Physiology - Wiley Online Library

Enhanced innate immune suppression by SARS-CoV-2 Omicron subvariants BA.4 and BA.5

How does SARS-CoV-2 evade the immune defences?

A comprehensive SARS-CoV-2 and COVID-19 review, Part 1: Intracellular overdrive for SARS-CoV-2 infection

Pathophysiological involvement of host mitochondria in SARS-CoV-2 infection that causes COVID-19: a comprehensive evidential insight