How Zika Virus Evades Antiviral Response of Cells

Researchers have identified a mechanism by which Zika virus can escape detection by host cells, offering a new target for therapeutics.

CD8+ T cells mediate protection against Zika virus induced by an NS3-based vaccine

Zika virus NS3 and NS5 proteins determine strain-dependent differences in dsRNA accumulation in a host cell type-dependent manner

Targeting Type I Interferon Induction and Signaling: How Zika Virus Escapes from Host Innate Immunity

STING ~ ViralZone

TLR3 Activation by Zika Virus Stimulates Inflammatory Cytokine Production Which Dampens the Antiviral Response Induced by RIG-I-Like Receptors

Zika virus employs the host antiviral RNase L protein to support replication factory assembly

Insights into the ZIKV NS1 Virology from Different Strains through a Fine Analysis of Physicochemical Properties

Zika virus antagonizes interferon response in patients and disrupts RIG-I–MAVS interaction through its CARD-TM domains, Cell & Bioscience

The innate immune response in Zika virus infection - Rodrigues de Sousa - 2021 - Reviews in Medical Virology - Wiley Online Library

Pathogens, Free Full-Text

Frontiers Neuroimmune Evasion of Zika Virus to Facilitate Viral Pathogenesis

What We're Doing to Combat the Zika Virus University of Washington - Department of Global Health