Cells, Free Full-Text
Por um escritor misterioso
Last updated 06 julho 2024
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Beyond their role in hemostasis, platelets have emerged as key contributors in the immune response; accordingly, the occurrence of thrombocytopenia during sepsis/septic shock is a well-known risk factor of mortality and a marker of disease severity. Recently, some studies elucidated that the response of platelets to infections goes beyond a simple fall in platelets count; indeed, sepsis-induced thrombocytopenia can be associated with—or even anticipated by—several changes, including an altered morphological pattern, receptor expression and aggregation. Of note, alterations in platelet function and morphology can occur even with a normal platelet count and can modify, depending on the nature of the pathogen, the pattern of host response and the severity of the infection. The purpose of this review is to give an overview on the pathophysiological interaction between platelets and pathogens, as well as the clinical consequences of platelet dysregulation. Furthermore, we try to clarify how understanding the nature of platelet dysregulation may help to optimize the therapeutic approach.
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Cell-Free DNA and Apoptosis: How Dead Cells Inform About the Living - ScienceDirect
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Polimer Program Get File - Colaboratory
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IJMS, Free Full-Text
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Remote immune processes revealed by immune-derived circulating cell-free DNA
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Antibodies, Free Full-Text
Mol Imaging Radionucl Ther on X: The Role of a Bone SPECT/CT Scan in the Follow-up of a Solitary Bone Lesion in a Patient with Langerhans' Cell Histiocytosis You can see the
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Experimental design of upsidedown, upside-up, and cell-free control
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Nucleic acid biomarkers of immune response and cell and tissue damage in children with COVID-19 and MIS-C - ScienceDirect
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IJMS, Free Full-Text
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Label-free, full-field visualization of red blood cell (RBC)
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Cancers, Free Full-Text
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Five-Year Outcomes for Refractory B-Cell Lymphomas with CAR T-Cell Therapy
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IJMS, Free Full-Text
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