CD209L/L-SIGN and CD209/DC-SIGN Act as Receptors for SARS-CoV-2
Por um escritor misterioso
Last updated 12 janeiro 2025
Biology, Free Full-Text
SARS CoV-2 spike protein variants exploit DC-SIGN/DC-SIGNR receptor for evolution and severity: an in-silico insight
Lectins enhance SARS-CoV-2 infection and influence neutralizing antibodies
Frontiers Depletion and Dysfunction of Dendritic Cells: Understanding SARS -CoV-2 Infection
Neuropilins: C-end rule peptides and their association with nociception and COVID-19 - Computational and Structural Biotechnology Journal
SARS CoV-2 spike protein variants exploit DC-SIGN/DC-SIGNR receptor for evolution and severity: an in-silico insight
DC/L-SIGN recognition of spike glycoprotein promotes SARS-CoV-2 trans-infection and can be inhibited by a glycomimetic antagonist
DC-SIGN and DC-SIGNR Interact with the Glycoprotein of Marburg Virus and the S Protein of Severe Acute Respiratory Syndrome Coronavirus
Human Cell Receptors and Downstream Cascades: A Review of Molecular Aspects and Potential Therapeutic Targets in COVID-19, Archives of Pediatric Infectious Diseases
CD209L (L-SIGN) is a receptor for severe acute respiratory syndrome coronavirus
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